Pulsed Electromagnetic Fields Stimulate HIF-1?-Independent VEGF Release in 1321N1 Human Astrocytes Protecting Neuron-like SH-SY5Y Cells from Oxygen-Glucose Deprivation

Disclaimer: this research study’s copyright belongs to the authors and PubMed and not HealthMatReview.com, further details below.

Our Summary

With PEMF emerging as one of the non-invasive therapeutic options for cerebral ischemia, this study aims to evaluate the effects of PEMF on human astrocytes, which plays an important role right after ischemia. Results showed that after PEMF treatment, there was a significant reduction in oxygen-glucose deprivation, induced cell proliferation and viability decrease in the neuron-like cells SH-SY5Y that strengthens the potential of PEMF treatment for cerebral ischemia.

Pubmed Study Link

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7663527/

PDF Study Link

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7663527/pdf/ijms-21-08053.pdf

Study Categories

cardio/heart/bloodflow

Authors

[‘Fabrizio Vincenzi’, ‘Silvia Pasquini’, ‘Stefania Setti’, ‘Simona Salati’, ‘Ruggero Cadossi’, ‘Pier Andrea Borea’, ‘Katia Varani’]

PMID

33126773

PMCID

PMC7663527

Published Date

more study details

Frequency (Hz)

75

Frequency Range

High

Intensity (G)

15

Intensity Range

High

Waveform

unknown

PEMF/EMF/RF/LT

PEMF

Study Subject Types

human

Keywords Listed

pulsed electromagnetic fields, VEGF, astrocytes, astrocyte-conditioned medium

Added Descriptive Keywords

blood flow, brain, oxygen level, low oxygen level, nervous system, human

Disclaimer

All intellectual property and copyright of this research study sourced from PubMed, is the property of the authors and associates and is not the property of HealthMatReview.com. This research study has been included in our custom PEMF research database for the educational purposes of our readers. The on page summary, added descriptive keywords and other study details are copyright of HealthMatReview.com and were added to improve the search function of this custom research database.

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